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128 results found

Vascular expression of E-selectin is increased in estrogen-receptor-negative breast cancer: a role for tumor-cell-secreted interleukin-1 alpha

Publication: Journal Article

Nguyen, Corless, Kräling, Tran, Atha, Bischoff, and Barsky. 1997. “Vascular Expression of E-Selectin Is Increased in Estrogen-Receptor-Negative Breast Cancer: A Role for Tumor-Cell-Secreted Interleukin-1 Alpha”. Am J Pathol 150 (4): 1307-14.

Epsin deficiency promotes lymphangiogenesis through regulation of VEGFR3 degradation in diabetes

Publication: Journal Article

Wu, Hao, Ashiqur Rahman, Yunzhou Dong, Xiaolei Liu, Yang Lee, Aiyun Wen, Kim Ht To, et al. 2018. “Epsin Deficiency Promotes Lymphangiogenesis through Regulation of VEGFR3 Degradation in Diabetes”. J Clin Invest.

A “CRISPR” view of Sturge-Weber syndrome is coming into focus

News: November 06, 2017

Three-dimensional modeling and CRISPR-Cas9 gene editing technology are giving scientists a new view into Sturge-Weber syndrome, a rare congenital disorder that causes small blood vessels, called capillaries, to be malformed.

Human blood vessels grown in mouse offer hope to heart attack patients

News: July 18, 2008

Scientists have grown human blood vessels in a mouse for the first time using cells taken from an adult donor.

The Sturge-Weber Foundation Awards 2nd Lisa’s Research Fellowship Award to Colette Bichsel, P.D

News: August 15, 2017

Tuesday, August 15, 2017; Houston, Texas – The Sturge-Weber Foundation (SWF), a 501 (c)(3) nonprofit organization, has named Colette Bichsel, PhD of Boston Children’s Hospital the recipient of the 2nd Lisa’s Sturge-Weber Research Fellowship Award.

Somatic GNAQ Mutation is Enriched in Brain Endothelial Cells in Sturge-Weber Syndrome

Publication: Journal Article

Huang, Lan, Javier Couto, Anna Pinto, Sanda Alexandrescu, Joseph Madsen, Arin Greene, Mustafa Sahin, and Joyce Bischoff. 2017. “Somatic GNAQ Mutation Is Enriched in Brain Endothelial Cells in Sturge-Weber Syndrome”. Pediatr Neurol 67: 59-63.

Endothelial colony forming cells and mesenchymal progenitor cells form blood vessels and increase blood flow in ischemic muscle.

Publication: Journal Article

KT, Kang, Lin RZ, Kupperman D, Melero-Martin J, and Bischoff J. 2017. “Endothelial Colony Forming Cells and Mesenchymal Progenitor Cells Form Blood Vessels and Increase Blood Flow in Ischemic Muscle.”. Sci Rep.

Nakayama, Hironao, Lan Huang, Ryan Kelly, Clara Oudenaarden, Adelle Dagher, Nicole Hofmann, Marsha Moses, Joyce Bischoff, and Michael Klagsbrun. 2015. “Infantile Hemangioma-Derived Stem Cells and Endothelial Cells Are Inhibited by Class 3 Semaphorins”. Biochem Biophys Res Commun 464 (1): 126-32.

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Vascular expression of E-selectin is increased in estrogen-receptor-negative breast cancer: a role for tumor-cell-secreted interleukin-1 alpha

Epsin deficiency promotes lymphangiogenesis through regulation of VEGFR3 degradation in diabetes

A “CRISPR” view of Sturge-Weber syndrome is coming into focus

Human blood vessels grown in mouse offer hope to heart attack patients

The Sturge-Weber Foundation Awards 2nd Lisa’s Research Fellowship Award to Colette Bichsel, P.D

Somatic GNAQ Mutation is Enriched in Brain Endothelial Cells in Sturge-Weber Syndrome

Endothelial colony forming cells and mesenchymal progenitor cells form blood vessels and increase blood flow in ischemic muscle.

Somatic Activating Mutations in GNAQ and GNA11 Are Associated with Congenital Hemangioma

Endoglin regulates mural cell adhesion in the circulatory system

Infantile hemangioma-derived stem cells and endothelial cells are inhibited by class 3 semaphorins