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Casein kinase I and casein kinase II differentially regulate axin function in Wnt and JNK pathways.

Publication: Journal Article

Zhang, Yi, Wen-Jie Qiu, Siu Chiu Chan, Jiahuai Han, Xi He, and Sheng-Cai Lin. 2002. “Casein Kinase I and Casein Kinase II Differentially Regulate Axin Function in Wnt and JNK Pathways.”. The Journal of Biological Chemistry 277 (20): 17706-12.

Destabilization of beta-catenin by mutations in presenilin-1 potentiates neuronal apoptosis.

Publication: Journal Article

Zhang, Z, H Hartmann, V M Do, D Abramowski, C Sturchler-Pierrat, M Staufenbiel, B Sommer, et al. 1998. “Destabilization of Beta-Catenin by Mutations in Presenilin-1 Potentiates Neuronal Apoptosis.”. Nature 395 (6703): 698-702.

SQI gallery contest

News: June 02, 2020

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Xi He, PhD

Person: Endowed Research Chair, American Cancer Society Research Professor

xi.he@childrens.harvard.edu

Mengxu Ge, PhD

Person: Postdoctoral Fellow

Studying the Wnt signaling in the skin. Mengxu.Ge@childrens.harvard.edu

Danilo Predes, PhD

Person: Postdoctoral Fellow

Studying the Wnt signaling in disease. Danilo.Predes@childrens.harvard.edu

Eunice To, PhD

Person: Postdoctoral Fellow

Studying the Atopic March. Eunice.To@childrens.harvard.edu

A Neurodegeneration Checkpoint Mediated by REST Protects Against the Onset of Alzheimer’s Disease

Publication: Journal Article

Aron*, Liviu, Chenxi Qiu*, Zhen Kai Ngian, Marianna Liang, Derek Drake, Jaejoon Choi, Marty A. Fernandez, et al. 2023. “A Neurodegeneration Checkpoint Mediated by REST Protects Against the Onset of Alzheimer’s Disease”. Nature Communications 14 (1).

Quantification of sulcal emergence timing and its variability in early fetal life: Hemispheric asymmetry and sex difference.

Publication: Journal Article

Yun, Hyuk Jin, Hyun Ju Lee, Joo Young Lee, Tomo Tarui, Caitlin K Rollins, Cynthia M Ortinau, Henry A Feldman, Ellen Grant, and Kiho Im. 2022. “Quantification of Sulcal Emergence Timing and Its Variability in Early Fetal Life: Hemispheric Asymmetry and Sex Difference.”. NeuroImage 263: 119629.

Loss of non-motor kinesin KIF26A causes congenital brain malformations via dysregulated neuronal migration and axonal growth as well as apoptosis.

Publication: Journal Article

Qian, Xuyu, Ellen M DeGennaro, Maya Talukdar, Shyam K Akula, Abbe Lai, Diane D Shao, Dilenny Gonzalez, et al. 2022. “Loss of Non-Motor Kinesin KIF26A Causes Congenital Brain Malformations via Dysregulated Neuronal Migration and Axonal Growth As Well As Apoptosis.”. Developmental Cell 57 (20): 2381-2396.e13.